Your brain on fast-acting carbs

Fast-acting carbs – like regular bread – light up the addiction centers in your brain like a Christmas tree: Your Brain on Fast Acting Carbs

To avoid this problem, eat mainly slow-acting unprocessed carbs, like vegetables. Plus protein and fat. Also called real food.


What Is Sugar Addiction? – Bitten Jonsson


Are You Addicted to Sugar?

Police Officer Losing Weight and Sugar Addiction with LCHF

“The Best 84 Days of My Life”

Ask Our Expert About Food Addiction

How to Get Free from Food Addiction



  1. Paul TR
    Well, I prefer to reduce carbohydrate intake rather than taking metformin
    Reply: #2
  2. murray
    Paul, speaking of low-carb and anti-aging, have you heard Dr. Ron Rosedale speak on mTor, surplus carbs/protein and the implications?

    (BTW, is there some way I can communicate directly with you.)

    Reply: #3
  3. Paul TR
    Hi Murray, thank you for the suggestion, yes I listened to dr Rosdale talk, although personally I have slightly different angle. As I mentioned on this blog numerous times, I do not care what people ate yesterday, 1 year, 10, 1,000, 1 million or 20 million year ago, I look how my body resound to nutrients now. Sending you a link to a paper about ketones and mTOR.
    Absolutely, let's communicate directly, maybe Dietdoctor can give us our respective e-mails incognito?.

    Epilepsia. 2011 Mar;52(3):e7-11. doi: 10.1111/j.1528-1167.2011.02981.x. Epub 2011 Mar 3.
    The ketogenic diet inhibits the mammalian target of rapamycin (mTOR) pathway.
    McDaniel SS1, Rensing NR, Thio LL, Yamada KA, Wong M.
    Author information

    The ketogenic diet (KD) is an effective treatment for epilepsy, but its mechanisms of action are poorly understood. We investigated the hypothesis that the KD inhibits mammalian target of rapamycin (mTOR) pathway signaling. The expression of pS6 and pAkt, markers of mTOR pathway activation, was reduced in hippocampus and liver of rats fed KD. In the kainate model of epilepsy, KD blocked the hippocampal pS6 elevation that occurs after status epilepticus. Because mTOR signaling has been implicated in epileptogenesis, these results suggest that the KD may have anticonvulsant or antiepileptogenic actions via mTOR pathway inhibition.

    Reply: #4
  4. murray

    I take Dr. Rosedale's invocation of evolution as more big-picture heuristic and rhetoric.

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