Talk about reversal of type 2 diabetes – not just disease management

April 18 2017 by in Diabetes, Dr. Sarah Hallberg

close up of man checking blood sugar by glucometer

Viewing type 2 diabetes as a chronic disease is quite outdated. Instead, we need a paradigm shift in accordance with the latest science, where we start recognizing that it is a highly reversible disease.

Here’s a great article from Dr. Sarah Hallberg:

With the increasing cost of health care, including $1 of every $3 in Medicare going to the treatment of T2D and its comorbidities, we have to look for solutions. In doing so, we must be willing to acknowledge that there have been past shortcomings in both dietary recommendations and treatment goals. Our patients deserve the opportunity to gain control of their health. They want more than just another prescription or procedure. To help them, we need to change the dialogue. We need to talk about reversal and provide the knowledge and support to achieve it.

AJMC: Type 2 Diabetes: Changing the Paradigm From Management to Reversal

How to Reverse Type 2 Diabetes

Earlier

NYT: Losing Weight and Reversing Diabetes with Virta Health

BMJ: Practices Should Be Able to Keep Money Saved from Putting Patients on a Low-Carbohydrate Diet

Video about type 2 diabetes with Dr. Sarah Hallberg

Reversing Diabetes by Ignoring the Guidelines – Dr. Sarah Hallberg

Video course about type 2 diabetes reversal

How to Reverse Diabetes Type 2 – Dr. Jason Fung

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The Essential Problem with Type 2 Diabetes - Dr. Jason Fung

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One comment

1
J Lance De Foa, MD
April 19 2017
The next step in the paradigm shift is taking the focus off the clinical sign of hyperglycemia with or without glycosuria (a.k.a. Diabetes mellitus, in Greek +Latin, or "pissing honey" in English) to a pathophysiologic mechanism focus, which is the secondary (diet induced) chronic hyperinsulinemia, and the Insulin-Toxicosis that it produces.

In tissues that normally respond to insulin surges we see "insulin resistance", and cortisol induced glucose release goes unchecked, or skeletal muscle which should take up even more glucose with insulin, does not.

Other tissues which normally ignore low levels of insulin become affected by it. Kidneys retain sodium and irate and engage in gluconeogenesis, and spill protein. Intraoccular pressure increases. White blood cells take to storing cholesterol in atheromas.

Just like primary hyperinsulinemia, known as insulinoma, patients can't help but increase storage of fat, just as those with aninsulinemia can't store fat without insulin.

In the late stage, just as thyrotoxicosis often burns itself out and results in Hypothyroidism, so exhausted beta-cells may eventually cease insulin production and patients become truly insulin dependent.

T2DM is Insulin-Toxicosis.